alternative medicine

Page 5 of 512345

Hypothesis hypothesis, they fed adult mice a high fat diet to induce obesity, steatosis and insulin resistance seen similar to humans. They studied the mouse liver lymphocyte population by flow cytometry. The researchers injected mice with liver inflammation causing exogenous lipopolysaccarhide and examined serum and liver tissue of the sacrificed mice.

They found point outt. – Tregs gradually decreased during the administration of high-fat diet and at the end of the 8 weeks, the concentrations of less than half of the. In mice ate a normal diet The high-fat diet induced simple steatosis, and made the mice more susceptible to LPS. Those on the high fat diet, the Tregs were depleted, suffered major damage to the liver, after having been exposed to LPS. ‘To our knowledge this is the first time that Tregs were the inflammatory process brought in the diet-induced steatosis in combination, ‘the authors report Interestingly, they also found that the Treg depletion reversed the mice were the mice were a high fat a high fat to a normal fat diet. Continue reading

Page 5 of 512345