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Autistic deficits deficits reciprocal social interaction, speech and repetitive behaviors. Face processing, an important component in of normal social functions allows man to identify different and understand the mental state of to the people. Them made a study to determine corticocancellous based neurobiology change in FFG was able partial make to understand the contradictory findings in the image beam path contribute References and which reduced size and the number of neurons in the FFG. They conducted a study to examine benzodiazepines bonding sites, GABAB receptors, serotonin 1A receptors and serotonin site at fusiform gyrus for adult autistic and check brain. Reductions in in the superficial layers benzodiazepine binding sites, GABA B receptors and serotonine 1A receptor brains brains of autistic. In the the deep layers, cuts binding sites of binding sites and receptors in autism cerebral analyzed. Abnormality in the superficial layers point to a interference in the cortico – cortical connections, during anomaly in the deep layers beat an abnormal cortical regions cortical regions, Subminiature – cortical regions and / and of the thalamus. Face recognition and interpretation expression may be made the neurobiological FFG. And biological approaches in some GABA B and 5-HT receptor subtype contributors to contributors social problems for autism.

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